Increasing levels of the mitofusin-1 (MFN1) protein in nerve cells of the spinal cord can reverse the features of Charcot-Marie-Tooth type 2A (CMT2A), a study in mice shows. The study, “Restoring mitofusin balance prevents axonal degeneration in a Charcot-Marie-Tooth type 2A model,” was published at the Journal of Clinical Investigation. CMT2A is caused by mutations … Continue reading Increased Levels of MFN1 Protein Can Reverse Features of CMT2A in Mice, Study Shows